Duplicating Dr. Rife's Cancer Treatment Success

By

James E. Bare, D.C.  

August 2026

 

It is unfortunate that Dr. Rife's results with cancer treatment cannot be fully duplicated today. In our present era, the use of frequencies to treat cancer is becoming ever more common . Unfortunately, results and treatment outcomes can vary significantly . Sometimes there is a remarkable response, sometimes there is no response, and all to often, there is a partial response.  Regardless of degree, responses can be temporary - a remission .  I have been focused upon trying to solve this enigma for over 30 years . A lot of time has been spent sorting out the urban legends and misinformation that abounds about Rife's results with cancer. What I have found is that the solution to achieving Dr. Rife's results lies not just in the device or it's frequency range capability. These qualities are highly important, but are not the full solution.   Modern plasma based frequency instruments have proven their efficacy over the decades.  Devices are tools that can enhance effects and outcomes,  and initiate the process of cancer cell death. Yes, for a  variety of reasons, some are better at this than others, but those reasons are beyond the scope of this document.  Before we are able to achieve Dr. Rife's level of treatment success, there are remaining mysteries that need to be  solved  . Recently, I've come to a much greater understanding of Dr. Rife's treatment method and the mechanisms involved.  From that understanding , the limitations we presently have that need to be overcome become obvious. This also has opened a door to a potential work around that may achieve close to the same level of effectivness.  The following information discusses Dr. Rife's methods, and provides insights into the incredible results achieved by Dr. Rife.

One portion of Dr. Rife's results with cancer came from eliminating the infectious bacillus component ( BX or BY) he discovered .  Dr. Rife focused not upon treating cancer, but rather destroying the organism he felt caused cancer using that organisms MOR. But, we now know, several different micro organisms and viruses are linked to the origin and or maintenance of many cancers, not just Rifes' BX or BY. Some viruses and micro organisms are cofactors in the origin of cancers. Others don't cause cancer, but activate processes within cancer cells that keep them growing and help to provide protective mechanisms.   In other words if a person has a cancer type known to be associated with certain micro organisms,  then in order to assist in the replication of Dr. Rife's effects,  it would be prudent to use  frequencies for those microorganisms and not just for BX or BY.  

Another portion of Dr. Rife's results, is based upon induction of irreversible cell death mechanisms/cascades which I mentioned in an earlier posting . Treatment with his BX or BY MOR frequency,  initiated a cell death mechanism. Dr. Rifes 1934 device trials had a 100% response rate, and all 16 patients were pronounced cured based upon the diagnostic criteria of the day. These days, the terms used are " complete response to treatment" and "remission" . One has to remember that this was the 1930's, a time where there were some basic blood tests, X-rays, and physical diagnosis to determine response to treatment. There were no CAT, MRI, or Scintillation scans. There were none of the highly sensitive blood tests that are now available. 

Dr. Rife's protocol was to do a very short  exposure of just three minutes,  and then do another exposure in two days .  Somehow, within those three minutes of frequency treatment,  programmed cell death mechanisms were activated, and cells died. Novocure has definitely proven that cancer cell death mechanisms occur from application of their Tumor Treatment (TT) fields. Modern devices ( including Novocure's) however take much longer to create activation of cell death mechanisms. Typically hours,  of exposure are required. When doing laboratory testing, Novocure prevents cell repair mechanisms from being able to salvage a damaged cancer cell  by applying their device 24 hours every day for 3 to five days. Their patients are encouraged to use their  device for as many hours a day as possible. Another way to look at this method is that when the cells are damaged by a frequency treatment, Novocure isn't allowing the cells to recover by taking a long pause between each treatment. 3 days is 72  hours, and that  is 4320 minutes, a long way from the 6 minutes exposure as done by Dr. Rife over the same time period !

Multiple papers that our research group have written, report on the cell killing effects of OPEF ( Oscillating Pulsed Electric Fields ) using 10 to 12 hour long exposures. As this testing is invitro ( culture plate), there is no immune system present that would otherwise attack damaged cancer cells.  The influence of the immune system should not be ignored in actual invivo treatment outcomes ! Part of the proposed work around process specifically enlists the immune system !

People tend not to use such prolonged exposure sessions when doing self treatment. An hour or two at one session is typical. Yet, many still improve using much shorter treatment times. To me that means that a significant amount of cancer cell damage occurs during each self treatment, and some of the cells are completing their death process.  By doing daily exposures,  the cells that did not die from the previous exposure do not have a chance to fully recover. 

Besides the patients immune systems, assisting in the patients treatment response,   I do believe  Dr. Rife's results came from some additional feature of the applied  frequency. A feature such as mixing of frequencies, gating, creation of beat frequencies,   or a combination of all of these. The output signal of Dr. Rifes instrument enhanced the initiation of cell death mechanisms that would occur from just a 3 minute exposure.  This topic is presently being explored by Dan Jensen using  Dr. Rife's original Kennedy receiver components.  Dr. Anthony Holland has developed a method of simultaneous multiple frequency generation known as   “Destructive Cancer Resonant Frequency Formant " ( DCRFF) . DCRFF  has shown significantly enhanced cell killing effects  when compared to the use of a single frequency upon leukemia cells. This is reported upon in the linked paper. https://www.preprints.org/manuscript/202305.2053

The cancer killing effects originated by Dr. Rife and his device,  have definitely been proven to exist.  The evidence is overwhelming that treatment with specific frequencies, and EM fields of adequate strength, can kill cancer cells. In fact the FDA has approved two devices I know of ,  the Novocure - "Optune" and the Therabionics "P1".  Society continues to suffer from the ravages of cancer through the disregard of this reality.  It's active suppression, is the safe haven domain of the scientific Luddites who refuse to acknowledge or fund research into these effects.

Sequential treatments by Dr. Rife were somehow able to avoid development of permanent cell protection and treatment resistance mechanisms.  I don't care how the cells died, I do know the cancer cells died using one or more of the 5 genetically pre programed cell death mechanisms. These are apoptosis, necroptosis, ferroptosis, autophagy, and pyroptosis.   The treatment process took 90 days for most of the group  to be declared as "cured" and 120 days for all of the group of patients to be declared as "cured". Looked at another way , 40 to 53 exposure treatments were required.  Each exposure treatment produced a roughly 2.25% reduction in the number of cancer cells by the time of the next exposure. 

A single exposure was not a " knock out punch", and for some reason, was not intended to be. Response to each short treatment was gradual and measured.  X-rays were used to treat cancer in Dr. Rife's time, and treatment resistance of cancer cells to X-rays following initial treatment was well known . Perhaps this acquired resistance effect had something to do with the protocol that was used ? There are obvious questions as to why the exposure was kept so short, and why exposures were done every 48 hours. What is known, is that this treatment regimen was effective.

What occurred after each exposure required several events; 1. that cancer cells died,  2. that cell division was slowed or inhibited in some manner, 3. that there always were fewer cancer cells present at the next exposure than the one prior, and 4. treatment resistance mechanisms were not activated.  Some understanding - cancer cells tend to divide on an average of 48 hours. Exposures were every 48 hours. One can see how it would be possible to have continued cell/tumor growth with this time duration between treatments.  Many of the cells killed by the exposure would  be replaced,  and most likely more cells than originally present would grow. But that did not occur ! There was a continuous, roughly  2.5% reduction in cancer cells from one exposure to the next.

It is important to raise this question " Did the cancer cells die simply because the bacillus Rife targeted was destroyed, or did this happen because the cancer cells were damaged by his applied frequency field for the bacillus ? Most likely, Dr. Rife's results were a consequence of both actions. 

One aspect that is often overlooked, is what happened long term to the patients that were treated?  Turns out, the issue of an incomplete cancer cell death process did affect Dr. Rife's long term results in the famous 1934 device trials. To quote " We also know that there was possibly two of those cancer patients who came back to Dr. Johnson with a recurrence of their cancer. " https://rifevideos.com/the_1934_cancer_and_tuberculosis_clinic.html "

This information is highly important, as it seems Dr. Rife was able to eliminate enough cancer cells to obtain a "cure" for the standards of his day, and not all the patients relapsed. Based upon available information, the majority in fact did not relapse ! For some reason, his treatment was not permanently activating cyto protective mechanisms including treatment resistance in the cancer cells that were not eliminated by his treatment. Was this due to the short exposure time of just 3 minutes combined with 2 days between treatments?

Cancer cells have enhanced DNA repair mechanisms as compared to normal cells. This especially so , within cancer stem cells. Besides the repair mechanisms, that are activated in response to attempts to kill cancer cells, also activated, are treatment resistance mechanisms.  In cancer stem cells, and surviving regular cancer cells, this activation is responsible for cancer tumor reoccurrence,  metastatic tumor spread, and treatment resistance. Worst of all, in response to repeated attacks such as from chemotherapy and radiation , and yes, even from frequencies,  cancer cells undergo genetic  alterations that makes the resistance mechanisms permanent.  Their resistance and repair mechanisms do not turn off,  and it becomes extremely difficult, if not impossible, to kill these cancer cells .

Here then are some of the critical questions and issues that needs solving so that we can obtain and then hopefully exceed Dr. Rife's results .

1. How does one activate Cell Death mechanisms using a RF initiated plasma with just three minutes of exposure ?

2. What is the role of frequencies and frequency ranges in response ?  This would include testing for effects using mixed types of frequencies in cell death initiation ? Dr. Rife used MHz level frequencies, TT field frequencies are in the 100Khz to 400 Khz regions, our research group is using from 80 to 200 Khz presently, Therabionics utilizes audio range frequencies for cancer treatment.

3. Does such cell death mechanism activation continue so the cells die ?

4. Does growth inhibition occur ?

5. How does elimination of any infectious component affecting cancer cell growth and replication affect the cells ?

6.  The total number of cells present in 48 hours needs to be lower than that of  48 hours prior.

7. There needs to be continued  reduction of cancer cell numbers, shrinkage of tumors, and a near total response to treatment  over 90 to 120 days of continued treatment

8. What must be done to assure cancer cells do not activate repair/develop resistance mechanisms, survive each treatment,  and continue to replicate after 90 to 120 days ?

9. What must be done to prevent genetic alteration so that the cancer cells do not develop permanent cytoprotective and treatment resistance mechanisms resulting in permanent treatment resistance ?

10. What happens with just 3 minutes of exposure time ? Can this be replicated?

Presently the answers to these questions and many others require laboratory research. I would estimate that a funded lab with a few scientists working in it could have answers to many of these questions inside of 6 months, and all of them within 18 months. Perhaps 2 million dollars to buy equipment, pay salaries and overhead,   and then recreate the treatment outcomes from nearly a century ago. Trying to do this work at an existing lab or higher education institution would be a fools errand.  Far to many conflicts of interest are found within the status quo.  Careers are at stake, political alignments exist, sensitive ego's are everywhere,  and gigantic amounts of money are on the line.  Recreating Dr. Rife's effective outcomes would destroy the status quo.  The total summation of the changes which would ensue would potentially place the lives of such researchers in jeopardy . The lab would have to be created from scratch using private funding.  But that isn't going to happen anytime soon....... 

There is a way to around this conundrum - we, that is, those of us within the frequency device community must solve the problem ourselves.  Either with crowd funded labs,  or with empirical reported results for feedback. The  solution  to cancer and replication of Dr. Rife's results, will never originate from some main stream research laboratory, a pharmaceutical company,  or school of higher education. They have had over 90 some years now to do this,  and they have not, and will not do this. 

Certainly Dr. Rife did not know the answers. He just had a method that worked!  A method of treatment that circumvented all the issues we presently are trying to work with . I believe it is possible  to use a "work around" to obtain improved treatment outcomes without directly knowing the answer of the unknowns surrounding Dr. Rife's results ! A nontoxic based protocol is  all that is required.  Cumulatively the number of ways this proposed protocol affects cancer cells is superior to that of Dr. Rife. But, the outcomes from this theorized protocol may or may not equal those of Dr. Rife.   I am hopeful that the outcomes from  his combined series of effects will result in consistent and reliable positive outcomes .  The protocol is driven by the use of frequency devices , and outcomes could be  better than any so far obtained by any other existing method.

To protect myself :  - the basic theorized protocol is a combination of existing known mechanisms and protocols that kill cancer cells, and that people are presently using. It is the emitted field of a frequency device, that will bind the mechanisms of these protocols together,  and provide the proof of my theory.  I must repeat - this is just a theoretical protocol. I am not diagnosing , treating , or actively encouraging those with cancer to utilize this method. This information is for educational purposes only.

As part of this theoretical protocol, it will be necessary that one have the medications already in their blood stream prior to using their frequency device.  People are already using these products and methods to treat cancers. But they are not combined, and used individually. It is the synergistic combination of the protocols which is all important.

Necessary components of my Treatment Theory:

1. A Frequency Device with adequate field strength and frequency range capability to initiate cell death mechanisms and open pores in the cancer cells that allows for significant improved intake of the medications . Novocure research has shown pores will open within cancer cells with fields as low as 1.5V/cm or 150 V per meter. These pores all the ingress of the assisting medications into the cancer cell.  Inhibition of mitotic spindle formation happens at 2.5 to 4 V per cm. Disrupting mitosis leads to failure to divide and cell death mechanisms being activated.  These are not high field strength values.  There are many devices which will easily provide these field strength levels.    Some of the devices are: Plasma Sonics - PGM-1 and PGM-2, Resonant Light PERL, GB-4000+MOPA, BCX, Hartwell DBx2 SSQ, True Rife, EMEM variants,  and others.

Here is a link to a paper on low voltage pore formation and enhanced medication uptake from late 2025 ( if using an anti tracker - it will need to be disabled) : https://aacrjournals.org/mct/article/24/11/1815/766858/Cancer-Cell-Permeability-Induced-by-Tumor-Treating

Besides pore formation and enhanced drug uptake , EM Fields have been shown to :

A. Distrupt the process of mitosis by upsetting spindle microtubule formation.

B. Cause Microtubule fragmentation during mitosis

C. Hinder DNA damage repair

D. Reduced metastasis - damage cell cytoskeleton

E. Activate autophagy and cell apoptois mechanisms.

2. Repurposed  Anthelmentics  protocols. People are using combinations of Ivermectin with Fenbendazole,  Ivermectin with  mebendazole. The results of studies using these products to kill cancer cells are quite positive and the internet is flooded with empirical outcome reports and suggested protocols for their use. All of these products need fat to be absorbed. Taken without fat - the absorption is miniscule and so are effects!  Typically it takes 6 months, or perhaps more, for significant effects to result from taking these medications.  Protocols are found on the web.

Mebendazole :

A. Inhibits microtubule polymerization during mitosis halting cell division

B. Switches macrophages cells into a cancer killing state , and activates T cells.

C. Induces DNA damage and autophagy

D. Inhibits enzymes that allow for metastasis and limits spread of cancer to organs.

E. Activates Apoptosis and autophagy . Importantly ( referring to quercetin actions)  inhibiting autophagy greatly enhances pro apoptosis and growth inhibition effects of Mebendazole.

Ivermectin :

A.  Distrupts mitochondrial function, which activates and enhances cell death via apoptosis

B Induces cell death via ferroptotis and autophagy.

C. Interferes with oncogenic pathways which leads to caspase activation -  which then leads to apoptotis and cell death.

D. "Decloakes" cancer cells from immune system allowing for immunogenic cell death  - promoting T cell infiltration, and reverses multi drug resistance mechanisms.

Fenbendazole:

A. Inhibits microtubule formation in mitosis, arrests mitosis, and this causes mitotic catastrophe.

B. Interferes with cancer cell enegy metabolism - inhibits glucose uptake

C. Activates apoptosis , pyroptosis, autophagy, p53 activation, and induces oxidative stress

D. Reduced angiogenesis - required for tumor growth

3. Quercetin : One must use Quercetin Phytosome with Bromelain for optimal absorption. Otherwise only about 17% of dose is absorbed. 500mg to 1000 mg/day is typical dose.


A. Is a radiosensitizer -  Term applied to ionizing radiation, these are also RF waves - just significantly higher in frequency.  Radioprotects normal cells from damage.

B. Inhibits DNA damage repair pathways. 

C. Activates and stabilizes p53 and  through this creates Apoptosis

D.  Activates mitochondrial  based apoptosis.

E. Inhibits metastasis, tumor invasion, and tumor proliferation.

F. Potentiates  the effects of anticancer drugs

G. Inhibits angiogenesis

H. Low doses of quercetin specifically inhibit cancer cell proliferation by inducing G1 phase cell cycle arrest, often via the upregulation of the p21 CDK

I. Activates caspases 3-8-9 based apoptosis  and promotes autophagy.

J. Slows tumor growth.


4. Treatment with known frequencies typically used to treat cancer. TT fields, Dr Rife's, public domain, and so on. Frequencies  derived via the method developed and patented by Charlene Boehm. Frequencies for infectious organisms associated with come cancers - example Epstein Barr Virus, HPV, HERV, HHV, MMTV, and so on. Just do a search for a particular cancer type of interest and see if there are viral or other infectious components to it's origin. Example " Breast Cancer Viral Links" .

 

Charlene Boehm's theorem can be used to calculate frequencies the could be used to reactivate various Tumor Suppressor genes that are turned off . These are not mutated genes, they are just turned off or inhibited in cancer cells.  These genes need to be turned back on.

One can also use RRM frequencies from Dr. Irina Cosic's theories

 

Some important Cancer Suppressor Genes and their frequencies

It is important that one check the action of these genes in the particular cancer type they are trying to work with. Some have very low presence, others can be close to 80%.  Formula used is from Ms. Boehm's patent

4526016.44/BP# = hz.   One can set the frequency range by multiplying by 2 to create octave harmonics. Use - 2,4,8,16,32,64,128,256,512 and so on ( 2^n ) this has been done for p53 as an example.

 

p53 Gene,  a major gene turned off in 50% of all cancers

20000bp = 452.601 hz     57933.010 hz   1853856.33 hz   3707712.66 hz

19143 bp = 472.863 hz    60526.574.hz   1936850.37 hz   3873700.74 hz

25760 bp =  702.797 hz    44979.045 hz  1439329.45 hz    2878658.903 hz 

 

PTEN Gene On average this gene is turned off in 13.5 % of all cancers. Is turned off in 65% of glioblastomas however.

100293 bp  =722.04 hz

 

CDKN2A Gene second most common gene turned off in cancers - about 30% - 40% of all cancers have this turned off .

is turned

27550 bp  = 657.1348 hz

23329 bp   = 776.03 hz

27573 bp   = 656.5867 hz

 

CDKN Gene

10870 bp  = 832.7537 hz

10957 bp = 826.141 hz

 

CDKN1B ( A.K.A. p27)

37876 bp = 955.96 hz

 

CDKN1C

2669 bp  = 1699.77 hz

2563 Bp = 1765.905 hz

 

p21 Gene

10879 Bp = 832.064 hz

 

PTCH1 Gene - inhibited in 50% of basal cell carcinomas ( most common skin cancer)

74078 Bp  = 977.5677

 

RBL2 Gene

57178 bp =633.252 hz

57673 bp = 627.8177hz

55744 bp = 649.543 hz


RBL1 Gene

99647 bp = 726.27 hz

100,000 bp = 724.162 hz

All of the medications are non toxic in regularly used dose levels, and frequency therapies have are almost no side effects.

 

Combined these methods create ( and this is not a complete list ) :

A. Disruption of Cancer Cell division - mitotic catastrophe occurs only in cancer cells with this protocol ! ( some common chemo drugs induce this - but damage normal cells as well ) 

B. Activation of Several different avenues of cell death without affecting normal healthy cells.

C. Inhibition of cancer cell growth, invasiveness,  and metastasis without the toxicity shown with chemotherapy drugs.

D. Activation of the immune system against cancer without damage to the immune system. Chemotherapy drugs often cause severe immune system damage.

E. Metabolic energy inhibitions (a common mode of action for many chemotherapy drugs ) affect only cancer cells, not normal cells.  Chemotherapy being non selective affects normal cells.  It is why people using ivermectin/mebendazole, fenbendazole or using frequency devices do not lose their hair  !

F. Tumor suppressor gene activation

G. The protocol is essentially non toxic.  Billions of doses of ivermectin/fenbendazole/ mebendazole have been taken over the decades with minimal intolerance issues.  The  extremely toxic and damaging side effects common to chemotherapy are not present and avoided.

There are other anthelmentics that might  be considered  besides the three mentioned above.  Some may eventually prove to provide superior effects with my proposed cancer treatment theory.

Here are the lists of anthelmentics either in clinical trial for cancer treatment or have shown anti cancer effects in the lab :

Anthelmintics undergoing clinical trials:

Albendazole

Ivermectin

Levamisole

Mebendazole

Niclosamide

Anthelmintics  having shown promising anti-cancer effects in the lab:

Flubendazole

Rafoxanide

Nitazoxanide

Praziquantel

 

It is my hope that this protocol will offer great benefit and just perhaps achieve close to the results of those obtained by Dr. Rife.

 

James E. Bare, D.C.

August 2026